Vanesa Gottifredi

Vanesa Gottifredi (born 13 November 1969) is an Argentine chemist and biologist. She works as a researcher in the Principal Investigator category of the Scientific and Technological Researcher Program (CICT) of the National Scientific and Technical Research Council (CONICET). She is also head of the Leloir Institute's Cell Cycle and Genomic Stability Laboratory. She specializes in the mechanisms of tumor cell response to chemotherapy, work for which she was awarded by the Alexander von Humboldt Foundation and L'Oreal-UNESCO. Provided by Wikipedia
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  1. 1
    The Tip of an Iceberg: Replication-Associated Functions of the Tumor Suppressor p53
    by Vanesa Gottifredi, Lisa Wiesmüller
    Published 2018-07-01
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  2. 2
    Current Understanding of RAD52 Functions: Fundamental and Therapeutic Insights
    by Vanesa Gottifredi, Lisa Wiesmüller
    Published 2020-03-01
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  3. 3
    Structural Chromosome Instability: Types, Origins, Consequences, and Therapeutic Opportunities
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  4. 4
    Under-Replicated DNA: The Byproduct of Large Genomes?
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  5. 5
    A monoclonal antibody against p53 cross-reacts with processing bodies.
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  6. 6
    CDK-Independent and PCNA-Dependent Functions of p21 in DNA Replication
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  7. 7
    Mitochondrial fusion is essential for steroid biosynthesis.
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  8. 8
    Chromosomal Integrity after UV Irradiation Requires FANCD2-Mediated Repair of Double Strand Breaks.
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  9. 9
    Persistent double strand break accumulation does not precede cell death in an Olaparib-sensitive BRCA-deficient colorectal cancer cell model
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  10. 10
    Cyclin Kinase-independent role of p21CDKN1A in the promotion of nascent DNA elongation in unstressed cells
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  11. 11
    The sesquiterpene lactone dehydroleucodine triggers senescence and apoptosis in association with accumulation of DNA damage markers.
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  12. 12
    Inhibitors of Rho kinases (ROCK) induce multiple mitotic defects and synthetic lethality in BRCA2-deficient cells
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  13. 13
    Correction: Inhibitors of Rho kinases (ROCK) induce multiple mitotic defects and synthetic lethality in BRCA2-deficient cells
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