Thomas C. Südhof

Südhof in 2024 Thomas Christian Südhof (; born December 22, 1955), ForMemRS, is a German-American biochemist known for his study of synaptic transmission. Currently, he is a professor in the school of medicine in the department of molecular and cellular physiology, and by courtesy in neurology, and in psychiatry and behavioral sciences at Stanford University.

Südhof, James Rothman and Randy Schekman are the 2013 Nobel Prize in Physiology or Medicine laureates for their work on vesicle trafficking.

Südhof retracted [https://www.pnas.org/doi/10.1073/pnas.2300363120 Lin et al. 2023] research paper published in PNAS from his lab due to falsified data, and since mid-2022, PubPeer commenters including Elisabeth Bik have flagged 46 of Südhof’s papers, which explore how neurons communicate across synapses. Provided by Wikipedia
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  1. 1
    Truth in Science Publishing: A Personal Perspective.
    by Thomas C Südhof
    Published 2016-08-01
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  2. 2
    The Perils of Navigating Activity-Dependent Alternative Splicing of Neurexins
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  3. 3
    LAR receptor phospho-tyrosine phosphatases regulate NMDA-receptor responses
    by Alessandra Sclip, Thomas C Südhof
    Published 2020-01-01
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  4. 4
    Latrophilin GPCR signaling mediates synapse formation
    by Richard Sando, Thomas C Südhof
    Published 2021-03-01
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  5. 5
    Combinatorial expression of neurexins and LAR-type phosphotyrosine phosphatase receptors instructs assembly of a cerebellar circuit
    by Alessandra Sclip, Thomas C. Südhof
    Published 2023-08-01
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  6. 6
    Cartography of teneurin and latrophilin expression reveals spatiotemporal axis heterogeneity in the mouse hippocampus during development.
    by Kif Liakath-Ali, Rebecca Refaee, Thomas C Südhof
    Published 2024-05-01
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  7. 7
    Latrophilin-2 and latrophilin-3 are redundantly essential for parallel-fiber synapse function in cerebellum
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  8. 8
    Neurexins regulate presynaptic GABAB-receptors at central synapses
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  9. 9
    Correction: Extended Synaptotagmin (ESyt) Triple Knock-Out Mice Are Viable and Fertile without Obvious Endoplasmic Reticulum Dysfunction.
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  10. 10
    Extended Synaptotagmin (ESyt) Triple Knock-Out Mice Are Viable and Fertile without Obvious Endoplasmic Reticulum Dysfunction.
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  11. 11
    Correction: Distinct neurexin-cerebellin complexes control AMPA- and NMDA-receptor responses in a circuit-dependent manner
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  12. 12
    Author Correction: Teneurins assemble into presynaptic nanoclusters that promote synapse formation via postsynaptic non-teneurin ligands
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  13. 13
    Teneurins assemble into presynaptic nanoclusters that promote synapse formation via postsynaptic non-teneurin ligands
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  14. 14
    Distinct neurexin-cerebellin complexes control AMPA- and NMDA-receptor responses in a circuit-dependent manner
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  15. 15
    A combinatorial code of neurexin-3 alternative splicing controls inhibitory synapses via a trans-synaptic dystroglycan signaling loop
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  16. 16
    Synaptotagmin-1 and -7 Are Redundantly Essential for Maintaining the Capacity of the Readily-Releasable Pool of Synaptic Vesicles.
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  17. 17
    RIBEYE B-Domain Is Essential for RIBEYE A-Domain Stability and Assembly of Synaptic Ribbons
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  18. 18
    Structural basis for a Munc13-1 homodimer to Munc13-1/RIM heterodimer switch.
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  19. 19
    Deletion of Calsyntenin-3, an atypical cadherin, suppresses inhibitory synapses but increases excitatory parallel-fiber synapses in cerebellum
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  20. 20
    A Trio of Active Zone Proteins Comprised of RIM-BPs, RIMs, and Munc13s Governs Neurotransmitter Release
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